Semax

Semax

Size:30mg
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Product details

Semax is a synthetic heptapeptide derived from the adrenocorticotropic hormone (ACTH) fragment ACTH(4-10). It is studied for its potential role in modulating neurochemical processes and influencing cellular pathways related to oxidative stress.

Key Properties

Neurochemical Research:

  • Investigated for its interaction with brain-derived neurotrophic factor (BDNF) and its receptor TrkB, which are linked to neuroplasticity.

Oxidative Stress Studies:

  • Explored for its potential to regulate cortical nitric oxide production and lipid peroxidation in laboratory models.

Neuroprotection Research:

  • Studied for its role in cellular viability and potential resistance to neurotoxic agents in controlled environments.

Applications in Research

Semax is a subject of ongoing laboratory research investigating:

  • Neurochemical pathways related to learning, memory, and neuronal survival.
  • Mechanisms of oxidative stress regulation and their impact on cellular function.
  • Cellular models focused on neuroprotection and resilience.

Storage and Handling Instructions

  • Store Semax in its lyophilized form at -4°F (-20°C) or lower.
  • Protect from light, moisture, and excessive heat.
  • Reconstitute peptides in sterile environments only.
  • Discard unused or reconstituted solutions promptly following research protocols.

Safety Information

Safety Information include:

  • Follow institutional safety guidelines during handling and disposal.
  • Use appropriate protective equipment.

Frequently Asked Questions

What is Semax?
Semax is a synthetic heptapeptide studied for its potential influence on neurochemical pathways, oxidative stress regulation, and cellular function.

How should Semax be stored?
Store Semax at -4°F (-20°C) in a sealed container protected from light and moisture. Reconstituted solutions should be used immediately following laboratory protocols.

What are the primary research focuses of Semax?
Research explores its effects on neurochemical pathways, oxidative stress mechanisms, and neuroprotection in controlled laboratory environments.

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